Artritis Gout, Diagnosis dan Pengelolaannya  

Oleh:

Iryaningrum, Maria Riastuti

Jenis: Article from Journal - ilmiah nasional - tidak terakreditasi DIKTI - atma jaya
Dalam koleksi: Majalah Kedokteran Damianus vol. 04 no. 03 (Sep. 2005), page 203-210.
Topik: ARTHRITIS; Hiperuricemia; Gouty Arthritis
Fulltext: D01 v4 n3 p203 kelik2023.pdf (986.82KB)

Ketersediaan

Perpustakaan PKPM Nomor Panggil: M61 Non-tandon: 1 (dapat dipinjam: 0) Tandon: tidak ada	Lihat Detail Induk
Perpustakaan FK Nomor Panggil: D01.K.01 Non-tandon: 1 (dapat dipinjam: 0) Tandon: tidak ada	Lihat Detail Induk

Isi artikel

Gout is a clinical syndrome, consisting of arthritis, tophi and nephrolithiasis, due 10 uric acid crystal accumulated on synovial, subcutaneous tissue, and renal tissue. This react for acute inflammation, and correlate with reduced renal filtration, tubular secretion and increased body reabsorbtion (primary gout) and accordingly with food consisting high purine, catabolic process due to medicine, and hormonal impairment such as hyperthyroid / hypthyroid, pre eclampsia. Gout started with hyperuricemia without clinical evidents (male > 7 mg%, female > 6 mg%) and then continued as acute gouty arthritis due to the accumulation of uric acids in the synovial tissue, then intercritical phase as a silent period from gouty attack and the chronical phase which manifest by the accumulated of uric acids out of synovial tissue. Management of gout (depends on stadium), is reducing Intake of food consisting uric acid (diet of low purine), consuming uricosuric and allopurinol as well.

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