Neuropathophysiology of Brain Injury  

Oleh:

Quillinan, Nidia ; Herson, Paco S. ; Traystman, Richard J.

Jenis: Article from Journal - ilmiah internasional
Dalam koleksi: Anesthesiology Clinics (keterangan: ada di ClinicalKey) vol. 34 no. 03 (Sep. 2016), page 453-464.
Topik: Mechanisms of Ischemic Brain Injury; Excitotoxicity; Oxidative Stress; Neuroinflammation; Apoptosis; Synaptic Plasticity; Neurovascular Unit; Translation
Fulltext: A34 v34 n3 p453 kelik2016.pdf (335.73KB)

Ketersediaan

Perpustakaan FK Nomor Panggil: A34.K Non-tandon: 1 (dapat dipinjam: 0) Tandon: tidak ada

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Isi artikel

Every year in the United States, millions of individuals incur ischemic brain injury from stroke, cardiac arrest, or traumatic brain injury. These acquired brain injuries can lead to death or long-term neurologic and neuropsychological impairments. The mechanisms of ischemic and traumatic brain injury that lead to these deficiencies result from a complex interplay of interdependent molecular pathways, including excitotoxicity, acidotoxicity, ionic imbalance, oxidative stress, inflammation, and apoptosis. This article reviews several mechanisms of brain injury and discusses recent developments. Although much is known from animal models of injury, it has been difficult to translate these effects to humans.

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