Detail Cor triatriatum sinister and cryptogenic stroke (artikel di dalam Jurnal Herz Cardiovascular Diseases, Vol. 40, Issue 3, Mei 2015, hal 447-448) Bibliografi Author: Ridjab, Denio Adrianus ; Wittchen, F. ; Tschishow, W ; Buddecke, J. ; Lamp, B. Topik: STROKE; cerebral infarctions; cryptogenic stroke; hypesthesia; cardiac embolism Bahasa: (ID )     Penerbit: Springer Link     Tahun Terbit: 2015     Jenis: Article - diterbitkan di jurnal ilmiah internasional Fulltext: 2015-jurnalinternasional-ridjab.pdf (241.91KB; 2 download) [Informasi yang berkaitan dengan koleksi ini di internet] Abstract Case report A 44-year-old woman with multiple cerebral infarctions was referred to our echocardiography laboratory from the neurology department for an echocardiogram in search of a cardioembolic source of her strokes. She complained of hypesthesia of her left hand on the ulnar side for the last 6 weeks. Her previous history was uneventful; cardiac symptoms were denied. Her heart rate was regular and normal as was her blood pressure (115/60 mmHg). The physical examination revealed a positive left Froment’s sign. She also had a sensory defect for touch sensation in the left fourth and fifth finger. Additionally, hypesthesia of the left forefoot was detected. There was no sign of heart failure; jugular veins were unremarkable. The resting electrocardiogram showed normal sinus rhythm; in addition, during a 24 h Holter recording, no atrial fibrillation or other significant arrhythmia was found. Ambulatory blood pressure monitoring showed normotensive values with normal circadian changes. Doppler examination of the neck arteries was normal without signs of atherosclerosis. The laboratory results were unremarkable, particularly screening for coagulopathy was negative (AT III, protein S, protein C, plasminogen, APC resistance, prothrombin II GA 20210 mutation, and anticardiolipin antibodies). Magnetic resonance imaging (MRI) of the neurocranium showed defects in the posterior parietal territory on both sides and a small defect in the right posterior inferior cerebellar artery territory. There was no sign of an acute ischemia and no sign of vascular abnormality. The transthoracic echocardiography performed in the search of a cardiac source of embolism was limited by a suboptimal acoustic window. Transesophageal echocardiography showed a membrane in the left atrium, incompletely dividing the atrium into two chambers (Fig. 1, Fig. 2, Fig. 3). No pressure gradient across the membrane was detected. Spontaneous echo contrast was not visible and the left atrial appendage was free of thrombus. The left atrial dimension was normal (volume 32 ml); right and left ventricular function was also normal. A patent foramen ovale was not visible; no shunt could be provoked or visualized after injection of agitated Gelafundin 4?%. Opini Anda Klik untuk menuliskan opini Anda tentang koleksi ini! Lihat Sejarah Pengadaan  Konversi Metadata   Kembali

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