Chronic Obstructive Pulmonary Disease: Aetiology, Pathology, Phsiology and Outcome  

Oleh:

Polkey, Michael I

Jenis: Article from Bulletin/Magazine - ilmiah internasional
Dalam koleksi: Medical Progress vol. 36 no. 01 (Jan. 2009), page 13.

Ketersediaan

Perpustakaan FK Nomor Panggil: M36.K.01 Non-tandon: 1 (dapat dipinjam: 0) Tandon: tidak ada

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Isi artikel

The Most common aetiology of chronic pulmonary disease (COPD ) is noxious practicles in the form of tobacco smoke, but the importance of other agents, particularly biomass fuels, is noted. Genetic dispositions are also relevant, and there in one known genetic condition, alpha-1-antitrypsin deficiency, which causes emphysema. Recurrent infections and inflammation are also important. Mucus hypersecretion and progressive airflow limitation, which causes hyperinflation, charaterize the condition. Exacerbations requiring admission to hospital are particularly dangeraous with an in-hospital mortality of 7% in aresent UK survey. Co-morbidities associated with COPD Include Cardiosvascuilar disease and osteoporosis. Direc secondary complications include cachexia, loss of fat-free mass and skeletal muscle weakness

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