On the Caffeination of Prematurity  

Oleh:

Stevenson, David K.

Jenis: Article from Journal - ilmiah internasional
Dalam koleksi: The New England Journal of Medicine (keterangan: ada di Proquest) vol. 357 no. 19 (Nov. 2007), page 1967.

Ketersediaan

Perpustakaan FK Nomor Panggil: N08.K.2007.06 Non-tandon: 1 (dapat dipinjam: 0) Tandon: tidak ada

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Isi artikel

It was recognized more than three decades ago1 that methylxanthines can reduce the frequency of apneic episodes in premature infants. Since that time, methylxanthines have become part of the routine clinical management of apnea of prematurity, with caffeine the treatment of choice because of its long half-life in the neonate.2 However, there has been ongoing concern, and a lack of good data, about potential long-term risks. In particular, caffeine can decrease cerebral blood flow in adults (when present at levels that are probably higher than the doses recommended for therapeutic use in babies)3 and can inhibit adenosine receptors,4 possibly compromising . . .

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