The Hemostatic System as a Modulator of Atherosclerosis  

Oleh:

Borissoff, Julian Ilcheff ; Spronk, Henri M.H. ; Cate, Hugo ten

Jenis: Article from Journal - ilmiah internasional
Dalam koleksi: The New England Journal of Medicine (keterangan: ada di Proquest) vol. 364 no. 18 (May 2011), page 1746-1760.
Topik: CARDIOVASCULAR DISEASES

Ketersediaan

Perpustakaan FK Nomor Panggil: N08.K.2011.02 Non-tandon: 1 (dapat dipinjam: 0) Tandon: tidak ada

Lihat Detail Induk

Isi artikel

Cardiovascular disease is one of the leading causes of death and complications worldwide. The classic concept of atherosclerosis assigns a pivotal role to inflammation in the onset and progression of this disease. Various inflammatory cell types (e.g., macrophages, neutrophils, and lymphocytes) play crucial roles in the destabilization and subsequent rupture or erosion of an atherosclerotic plaque, ultimately resulting in atherothrombosis. Inflammation is closely linked to coagulation in several pathologic conditions. Intriguingly, extensive bidirectional cross-talk between the two systems has been established in many complex diseases, including atherosclerosis.

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