Dissociation of Voluntary and Emotional Innervation after Stroke  

Oleh:

Kappos, Ludwig

Jenis: Article from Journal - ilmiah internasional
Dalam koleksi: The New England Journal of Medicine (keterangan: ada di Proquest) vol. 363 no. 16 (Nov. 2010), page 363:e25.
Topik: Foto Mimik Muka
Fulltext: Foto Mimik Muka.pdf (47.72KB)

Ketersediaan

Perpustakaan FK Nomor Panggil: N08.K.2010.01 Non-tandon: 1 (dapat dipinjam: 0) Tandon: tidak ada

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A 72-year-old man presented with a sudden onset of slurred speech. His medical history was unremarkable, and he was taking no medications and did not smoke. The physical examination revealed left-sided facial paralysis and dysarthrophonia. Diffusion-weighted magnetic resonance imaging showed a hyperintensity in the right precentral gyrus, reflecting acute cerebral ischemia (Panel A, arrow). Three days after the stroke, the patient showed impaired voluntary innervation. When he was asked to smile on command, he was unable to fully smile because of facial paralysis on the left side (Panel B). However, during emotional encounters, the patient was able to overcome the facial paralysis (Panel C). It is hypothesized that this dissociation of emotional and volitional facial movement is due to separate origins of corticofacial projections. The nerve tracts affecting voluntary facial movement probably originate from the main motor cortex. Those affecting involuntary movement during emotion probably arise from the caudal cingulate motor cortex, a medial brain region with inputs from the limbic system (see figure in the Supplementary Appendix).

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