Efek dari Kecanduan Alkohol Terhadap Peningkatan Kerusakan Hati  

Oleh:

Simanjuntak, Kristina

Jenis: Article from Journal - ilmiah nasional
Dalam koleksi: Bina Widya: Majalah ilmiah UPN "Veteran" Jakarta vol. 23 no. 1 (Oct. 2011), page 35-42.
Topik: Alcohol; Exposure; Free Radical; Liver Injury

Ketersediaan

Perpustakaan Pusat (Semanggi) Nomor Panggil: BB24.3 Non-tandon: 1 (dapat dipinjam: 0) Tandon: tidak ada

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Isi artikel

Alcohol drinking are favorite in are people. The like is drink of etanol are want to know taste, emotional and low pshycic. After alcohol drinking, in low level will be to influence happy taste, and then if consumed alcohol to high level actually they most talked, nausea, dizzing, tasty, blood pressure low. Alcohol dependent to psychological and social actually self no control. The characteristic of alcohol toxic in the body, because they are faster to damage of lever and brain tissue. The damage liver from alcoholism can be via induced enzyme alcohol dehydrogenase and high free radical level. The alcohol dehydrogenase enzyme can change alcohol to acetaldehyde and then finally to oxidized to acetat or acetil koA form. Acetil koA are first agent to lipogenesis process, and then to causing accumulation high lipid in liver, to formed hiperlipidimia, cirrhosis and finally to death. The react alcohol metabolism will be to free radical and react to macromolecule which causing stress oxidative cell.

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