Detail Forever Young; Ageing   Oleh:  [s.n] Jenis: Article from Bulletin/Magazine Dalam koleksi: The Economist (http://search.proquest.com/) vol. 401 no. 8758 (Nov. 2011), page 89-90. Topik: Ageing; Research; Cell Division Ketersediaan Perpustakaan Pusat (Semanggi) Nomor Panggil: EE29.68 Non-tandon: 1 (dapat dipinjam: 0) Tandon: tidak ada     Lihat Detail Induk Isi artikel A way to counteract part of the process of growing old Biologists have made a lot of progress in understanding ageing. They have not, however, been able to do much about slowing it down. Particular versions of certain genes have been shown to prolong life, but that is no help to those who do not have them. A piece of work reported in this week's Nature by Darren Baker of the Mayo Clinic, in Minnesota, though, describes an extraordinary result that points to a way the process might be ameliorated. Dr Baker has shown--in mice, at least--that ageing body cells not only suffer themselves, but also have adverse effects on otherwise healthy cells around them. More significantly, he has shown that if such ageing cells are selectively destroyed, these adverse effects go away. The story starts with an observation, made a few years ago, that senescent cells often produce a molecule called P16{+I}{+N}{+K}{+4}{+A}. Most body cells have an upper limit on the number of times they can divide--and thus multiply in number. P16{+I}{+N}{+K}{+4}{+A} is part of the control mechanism that brings cell division to a halt when this limit is reached. Opini Anda Klik untuk menuliskan opini Anda tentang koleksi ini! Kembali

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